Calcitonin: Difference between revisions
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Calcitonin is a 32 residue polypeptide hormone synthesized primarily in the thyroid and is able to decrease blood calcium levels by enhancement of calcium excretion from the kidney as well as direct inhibition of mediated bone reabsorption<ref>Masi, Laura, and Maria Luisa Brandi. “Calcitonin and Calcitonin Receptors.” Clinical Cases in Mineral and Bone Metabolism 4.2 (2007): 117–122. Print.</ref>. | Calcitonin is a 32 residue [[polypeptide hormone|polypeptide hormone]] synthesized primarily in the [[thyroid|thyroid]] and is able to decrease [[blood|blood]] [[calcium|calcium]] levels by enhancement of calcium excretion from the [[kidney|kidney]] as well as direct inhibition of mediated [[bone reabsorption|bone reabsorption]]<ref>Masi, Laura, and Maria Luisa Brandi. “Calcitonin and Calcitonin Receptors.” Clinical Cases in Mineral and Bone Metabolism 4.2 (2007): 117–122. Print.</ref>.<br> | ||
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=== References === | === References === | ||
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Latest revision as of 18:13, 16 October 2018
Calcitonin is a 32 residue polypeptide hormone synthesized primarily in the thyroid and is able to decrease blood calcium levels by enhancement of calcium excretion from the kidney as well as direct inhibition of mediated bone reabsorption[1].
References
- ↑ Masi, Laura, and Maria Luisa Brandi. “Calcitonin and Calcitonin Receptors.” Clinical Cases in Mineral and Bone Metabolism 4.2 (2007): 117–122. Print.