Islet Amyloidosis: Difference between revisions
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Human islet amyloid polypeptide or amylin (hA) is a 37-amino acid [[Peptide|peptide]] [[Hormone|hormone]] produced and co-secreted with [[Insulin|insulin by]] [[Pancreatic beta cells|pancreatic β-cells]]. Under physiological conditions, hA regulates a broad range of biological processes including insulin release and slowing of gastric emptying, thereby maintaining [[Glucose|glucose]] [[Homeostasis|homeostasis]]<ref>Handb Exp Pharmacol. 2017 Oct 18. doi: 10.1007/164_2017_62.</ref>. | Human islet amyloid polypeptide or amylin (hA) is a 37-amino acid [[Peptide|peptide]] [[Hormone|hormone]] produced and co-secreted with [[Insulin|insulin by]] [[Pancreatic beta cells|pancreatic β-cells]]. Under physiological conditions, hA regulates a broad range of biological processes including insulin release and slowing of gastric emptying, thereby maintaining [[Glucose|glucose]] [[Homeostasis|homeostasis]]<ref>Handb Exp Pharmacol. 2017 Oct 18. doi: 10.1007/164_2017_62.</ref>. | ||
=== References === | |||
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Latest revision as of 15:31, 19 October 2017
Human islet amyloid polypeptide or amylin (hA) is a 37-amino acid peptide hormone produced and co-secreted with insulin by pancreatic β-cells. Under physiological conditions, hA regulates a broad range of biological processes including insulin release and slowing of gastric emptying, thereby maintaining glucose homeostasis[1].
References
- ↑ Handb Exp Pharmacol. 2017 Oct 18. doi: 10.1007/164_2017_62.